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. 1999 Nov 29;147(5):995–1008. doi: 10.1083/jcb.147.5.995

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© 1999 The Rockefeller University Press

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 4.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/4.0/).

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Abnormal localization and reduced accumulation of IP3 receptors in cardiomyocytes and thymus of ankyrin-B (−/−) mice. Cardiomyocytes (A) and sections of thymus (B) of ankyrin-B (+/+) (left) and ankyrin-B (−/−) mice are labeled with antibody against IP3 receptor (types 1, 2, and 3) and visualized by immunofluorescence. Levels of label were computationally enhanced about twofold in ankyrin-B (−/−) examples to reveal localization. C shows immunoblots of equivalent amounts of ankyrin-B (+/+) and (−/−) thymus (left) and heart (right) with antibody against the IP3 receptor. Bars: (A) 10 μm; (B) 5 μm.