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RGS4 increased the amplitude of GTPγS-induced GIRK current. (A) In the absence of any exogenous receptors, RGS4 coexpression caused a larger GIRK current induced by GTPγS. (B and C) When μ-receptors were expressed and stimulated by DAMGO (B) or nalorphine (C), RGS4 coexpression drastically increased the amplitudes of the induced current but not the time to reach maximal activation.
