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. 2003 Oct 27;163(2):223–229. doi: 10.1083/jcb.200306075

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Copyright © 2003, The Rockefeller University Press

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Figure 4. — Retrograde axonal transport is disrupted in BPAG1 null mice. (A and B) EM of longitudinal sections of spinal cord dorsal column reveals accumulation of vesicles and other organelles in BPAG1 null (A), but not in WT (B). (C–E) Proteins failed to accumulate on the distal side of a double ligature (schematic shown in C) of BPAG1 null sciatic nerves. Shown are confocal images of nerve sections stained with anti-p75^NTR (D), and immunoblots of p75^NTR from ∼3-mm sections proximal or distal to double ligatures (E). Anti–α-tubulin served as loading control. Bar: (A and B) 500 nm; (D) 140 μm.