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. 1999 Dec 27;147(7):1365–1370. doi: 10.1083/jcb.147.7.1365

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© 1999 The Rockefeller University Press

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 4.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/4.0/).

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graphic file with name JCB9906002.f3a.jpg — The DNA binding function of the GR is dispensable for repression of MMP-13 and MMP-3 induction in vivo. Wild-type (wt) or GR^dim mice were treated with acetone (Co), 50 μg dexamethasone (Dex), 10 nmol TPA (TPA), or 10 nmol TPA with 50 μg dexamethasone (T+D). Animals were killed after 6 h (a) or 1.5 h (b) and analyzed by Northern blot.

graphic file with name JCB9906002.f3b.jpg — The DNA binding function of the GR is dispensable for repression of MMP-13 and MMP-3 induction in vivo. Wild-type (wt) or GR^dim mice were treated with acetone (Co), 50 μg dexamethasone (Dex), 10 nmol TPA (TPA), or 10 nmol TPA with 50 μg dexamethasone (T+D). Animals were killed after 6 h (a) or 1.5 h (b) and analyzed by Northern blot.