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. 2007 Oct 12;86(3):245–256. doi: 10.1007/s11060-007-9470-8

Table 1.

WIP1 amplification is associated with gain of chromosome 17q or i17q in primary medulloblastoma specimens

Specimen Copy number (FISH) Amplification* CNA (CGH)
CEP 17 WIP1 WIP1 Chr 17 status
D283 2–4 4–9 Y i17q
Daoy 4 3 N +17
MCF-7 4 >50 Y **
MB13 2 6–7 Y −17p12-p13, +17p11.2-q25
MB1 2–4 5–8 Y i17q
MB3 2–4 3–7 Y i17q
MB14 2–4 2–7 Y +17
MB15 2–4 2–6 Y +17
MB5 2 6 Y i17q
MB2 3–4 6–11 Y i17q
MB6 2 4 N i17q
MB28 2 2 N N/C
MB27 2 2 N N/C
MB4 2–3 2–3 N i17q

FISH analysis for WIP1 and chromosome 17 centromere reveals low-level amplification of WIP1 in 7 of 11 primary medulloblastoma samples. WIP1 copy gain is significantly associated with gain of 17q (P = 0.00057)

*Amplification is defined as >4 copies as determined by FISH

**Reportedly amplified, but not determined in the present study

Abbreviations: MB, medulloblastoma; +17, gain of chromosome 17; −17, loss of chromosome 17; N/C, no change in chromosome 17; CNA, copy number aberration; CEP 17, centromeric chromosome 17 probe