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. 1999 Jan 18;189(2):253–264. doi: 10.1084/jem.189.2.253

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The Vα2Vβ5-specific antagonist peptides E1 and P7 do not antagonize the response of the Vα2Vβ10 TCR to the Herpes simplex viral peptide, SSIEFARL. Vα2Vβ10 58CD8α/β hybridomas were stimulated with various concentrations of SSIEFARL in the presence of the Vα2Vβ5-specific antagonists, E1 and P7, or control peptide, K4. The IL-2 response (A) and antigen-induced TCR internalization (B) were measured in the same experiment and are representative of two independent experiments.