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. 2003 Dec 15;198(12):1785–1796. doi: 10.1084/jem.20021562

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Copyright © 2003, The Rockefeller University Press

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Figure 1. — NKT-deficient mice have defective CS. (A) CD1d^−/− mice (Group C) and WT CD1d^+/+ controls (Group A) were skin sensitized with 5% PCl and on day 4 were ear challenged with 0.4% PCl. Nonimmune CD1d^−/− and CD1d^+/+ (Groups D and B) controls were similarly ear challenged. Ear swelling was measured at 2 h (left) and 24 h (right). The results represent pooling from three different experiments each with three mice per group. (B) Jα18^−/− mice (Group C) and WT BALB/c controls (Group A) were sensitized and ear challenged. Nonimmune Jα18^−/− (Group D) and BALB/c (Group B) controls were ear challenged similarly. 2- and 24-h ear swelling responses were measured. The results are from a representative experiment with five mice per group.

Figure 1. — NKT-deficient mice have defective CS. (A) CD1d^−/− mice (Group C) and WT CD1d^+/+ controls (Group A) were skin sensitized with 5% PCl and on day 4 were ear challenged with 0.4% PCl. Nonimmune CD1d^−/− and CD1d^+/+ (Groups D and B) controls were similarly ear challenged. Ear swelling was measured at 2 h (left) and 24 h (right). The results represent pooling from three different experiments each with three mice per group. (B) Jα18^−/− mice (Group C) and WT BALB/c controls (Group A) were sensitized and ear challenged. Nonimmune Jα18^−/− (Group D) and BALB/c (Group B) controls were ear challenged similarly. 2- and 24-h ear swelling responses were measured. The results are from a representative experiment with five mice per group.