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. 1999 Oct 4;190(7):953–962. doi: 10.1084/jem.190.7.953

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© 1999 The Rockefeller University Press

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 4.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/4.0/).

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Cytokine production from T. muris–infected female BALB/c IL-4 KO mice after treatment with anti–TNF-α (black bars), control Ig (stippled bars), or PBS (striped bars). Mesenteric lymph node cells were removed on day 18 p.i., stimulated in vitro with 50 μg T. muris Ag for 24 h, and supernatants were analyzed by sandwich ELISA for the presence of IL-5 (A), IL-9 (B), and IL-13 (C). Results represent the mean values from four mice per group ± SEM. *Significantly lower than control treated groups (P < 0.05).

Cytokine production from T. muris–infected female BALB/c IL-4 KO mice after treatment with anti–TNF-α (black bars), control Ig (stippled bars), or PBS (striped bars). Mesenteric lymph node cells were removed on day 18 p.i., stimulated in vitro with 50 μg T. muris Ag for 24 h, and supernatants were analyzed by sandwich ELISA for the presence of IL-5 (A), IL-9 (B), and IL-13 (C). Results represent the mean values from four mice per group ± SEM. *Significantly lower than control treated groups (P < 0.05).