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Blood derived from mice deficient in the complement receptor CR3 demonstrates markedly reduced GBS-induced TNF-α release. Blood from CR3-deficient or age- and sex-matched control C57/129 mice was incubated with HK-GBS at 107 or 108 bacteria/ml before measurement of TNF-α by ELISA. Data represent the means ± standard errors of the means (n = 4 to 6). Differences between CR3-deficient and control mice were significant (P < 0.05, Fisher's exact test).
