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. 2002 Jan 7;195(1):71–84. doi: 10.1084/jem.2001889

Table II.

Histological Characterization of Inflammatory Lesions in DO11.RAG-2−/−/RAG-2−/− Recipients Colonized with E. coli-OVA versus E. coli-TET

T cells Intestinal colonization CD3+/mm2totala CD3+/mm2IEL PMN/mm2total Percentage of eosinophils
(PMNs)
Percentage of BrdU+(CD3)
Naive pnir15.TET-E. coli 77.4 ± 44.2b 5.5 ± 5.0 4.0 ± 3.7 ND 1.6 ± 1.7
pnir15.OVA-E. coli 192.3 ± 73.5 7.3 ± 3.6 8.5 ± 3.8 2.4 ± 2.9 24.0 ± 5.2
Th1 pnir15.TET-E. coli 203.7 ± 55.8 17.9 ± 8.4 17.9 ± 8.4 ND 2.5 ± 1.8
pnir15.OVA-E. coli 796.1 ± 190.3 66.5 ± 29.3 246.0 ± 97.3 5.7 ± 4.8 34.8 ± 7.6
Th1 pnir15.TET-E. coli 140.1 ± 50.0 13.0 ± 8.3 0.5 ± 1.0 ND 7.5 ± 1.9
pnir15.OVA-E. coli 385 ± 96.2 33.0 ± 5.2 496.7 ± 44.7 39.6 ± 8.9 38.8 ± 9.6

RAG-2−/− recipients were reconstituted with 106 CD4+ T cells of the indicated phenotypes that were isolated/generated from DO11.RAG-2−/− donors. 1 d before T cell reconstitution, recipients were colonized with 1010 CFU of the indicated bacteria and maintained with biweekly administration of an additional dose of 1010 CFU bacteria. 8 wk after reconstitution, mice were killed and necropsied.

a

The total number of CD3+ T cells per mm2 of colonic mucosa, including LP and IEL compartments (see Material and Methods for details).

b

Data are the means ± SEM from 3–6 animals per group. Values that differed significantly from control (P > 0.05) are indicated in bold font.

ND, not determined.