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. 1997 May 19;185(10):1769–1775. doi: 10.1084/jem.185.10.1769

Figure 4.

Figure 4

Ability of the anti-TGF-β (1D11.16) mAb to reverse LEWHgAmediated protection from mercury disease. Antilaminin antibody titer (A) and serum IgE concentration (B) in HgCl2-injected rats (□, n = 5), and HgCl2-injected rats 2 wk after adoptive transfer of the LEWHgA T cell line treated (▴, n = 4) or not (▪, n = 4) with the 1D11.16 anti-TGF-β mAb (2 mg at day −2, 0, 3, 6, and 9 with respect to the first HgCl2 injection). The result is a pool of two independent experiments.