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. 2008 Feb;178(2):947–956. doi: 10.1534/genetics.107.082115

TABLE 2.

Life span and neuropathology of double mutants with ATPαDTS1

50% ± SD Neuropathology 50% ± SD Neuropathology
Genotype +/+ +/+ DTS1/+ DTS1/+
+/Y 38 ± 4.4 0–1 14 ± 0.8 4
eas/Y 35 ± 2.8 2 4 ± 0.8 2
tko/Y 31 ± 2.3 1–3 3 ± 0.6 NA
sesB/Y 26 ± 1.8 3 7 ± 0.4 2
bss/Y 23 ± 5.7 0–1 5 ± 0.6 0–1
bas/Y 23 ± 3.5 1–2 4 ± 0.5 0
kdn/Y 18 ± 1.7 0–1 11 ± 2.8 0–1
+/+ 38 ± 4.4 0–1 23 ± 3.4 4
eas/+ 44 ± 3.1 1 14 ± 6.0 4
tko/+ 48 ± 4.6 1 16 ± 6.4 3–4
sesB/+ 43 ± 7.8 2 27 ± 3.2 3–4
bss/+ 45 ± 1.0 1 15 ± 3.8 3–4
bas/+ 46 ± 5.3 0 24 ± 7.4 3
kdn/+ 44 ± 3.5 0–1 20 ± 6.2 2–4

The life span of males hemizygous for a bang-sensitive mutation (top) in combination with DTS1 is very reduced in comparison with heterozygous bang-sensitive females (bottom), which may not allow sufficient time for neurodegeneration to appear grossly as vacuolar brain lesions. Longevity (at 29°) is presented as the age in days at which 50% of the flies of the indicated genotype have died. Third chromosome genotype is indicated along the top. DTS1 is ATPαDTS1, a dominant temperature-sensitive ATPα allele. Neuropathology was scored 0 (none) to 5 (marked) at the median age. NA, not available.