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. 2005 Oct;126(4):379–392. doi: 10.1085/jgp.200509355

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Copyright © 2005, The Rockefeller University Press

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Figure 9. — Effect of mutations in ITR-1, the IP₃ kinase LFE-2, and the IP₃ phosphatase IPP-5 on pBoc arrhythmia (quantified as coefficients of variance) induced by RNAi-mediated loss of EGL-8 or PLC-3 activity. lfe-2(sy326) and ipp-5(sy605) loss-of-function and itr-1(sy327) gain-of-function mutant worms were fed either plc-3 or egl-8 dsRNA. itr-1, lfe-2, and ipp-5 mutations fully suppress arrhythmia induced by plc-3 RNAi (see Fig. 5 B). Values are means ± SEM (n = 6–9). 10 pBoc cycles were measured for each worm. **, P < 0.001 compared with lfe-2 or lfe-2;plc-3(RNAi). *, P < 0.05 compared with ipp-5 or ipp-5;plc-3(RNAi). ^†, P < 0.001 compared with sy327 or sy327-2;plc-3(RNAi).