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. Author manuscript; available in PMC: 2008 Mar 16.
Published in final edited form as: Neurobiol Dis. 2007 Jan 19;26(1):14–26. doi: 10.1016/j.nbd.2006.11.014

Fig. 6.

Fig. 6

Normal PHR and amygdala anatomy in Fgf14−/− mice. (A) Normal hippocampal cytoarchitecture in the s.o., s.p. and s.r. of CA1 revealed by Nissl staining in Fgf14−/− and WT mice. (B–F) No difference was observed in the staining intensity of GFAP (B), SNAP-25 (C), Synaptophysin (Syn38) (D), PKCγ (E) and CaMKII (F) in CA1 regions of Fgf14−/− and WT mice. (G–H) Normal cytoarchitecture in the entorhinal cortex (G) and amygdala (H) in WT and Fgf14−/− mice (Nissl stain). BLA, anterior basolateral amygdaloid nucleus; BMA, anterior basolateral amygdala; LaDL, dorsolateral amygdaloid nucleus DEn, dorsal endopiriform nucleus; Pir, piriform cortex; rf, rhinal fissure; s.l-m., stratum lacunosum moleculare; s.o., stratum oriens; s.p., stratum pyramidal; s.r., stratum radiatum. Bar=125 µm (A, B); Bar=50 µm (C, D); Bar=500 µm (E–H).