. 2000 Jan 15;522(Pt 2):311–320. doi: 10.1111/j.1469-7793.2000.00311.x

Table 3.

Changes of working heart functions after infusion of DEA/NO or adenosine in the absence and presence of NOS inhibition

	CF (ml min⁻¹ g⁻¹)	CO (ml min⁻¹ g⁻¹)	LVP (mmHg)	+dP/dt_max (mmHg s⁻¹)	−dP/dt_max (mmHg s⁻¹)	Heart rate (beats min⁻¹)
Basal	9.9 ± 0.53	69.0 ± 5.5	83.8 ± 2.9	3089 ± 265	2989 ± 211	283 ± 15
DEA/NO	15.4 ± 0.62^*	73.1 ± 4.7^*	84.9 ± 2.6^*	3282 ± 223^*	3264 ± 247	295 ± 12
Basal	10.7 ± 0.45^*	70.0 ± 4.8^*	83.9 ± 2.1	3149 ± 213^*	3060 ± 199	275 ± 12^*
L-NA	7.0 ± 0.41^*	56.8 ± 3.6^*	80.4 ± 1.7^*	2792 ± 158^*	2470 ± 137^*	260 ± 10
L-NA +adenosine	13.8 ± 1.06^*	57.8 ± 4.8	81.1 ± 2.2	2938 ± 200	2510 ± 207	270 ± 13
L-NA	6.5 ± 0.38^*	49.0 ± 4.6^*	78.2 ± 2.2^*	2578 ± 168^*	2170 ± 133^*	258 ± 11
L-NA + DEA/NO	13.1 ± 0.45^*	70.1 ± 4.1^*	84.2 ± 2.2^*	3191 ± 192^*	2808 ± 175^*	282 ± 10^*

Values for coronary flow (CF), cardiac output (CO), left ventricular peak pressure (LVP), +dP/dt_max, −dP/dt_max and heart rate were measured at baseline and after infusion of 0.1 μm DEA/NO and 50 μm adenosine in the presence of the NOS inhibitor L-NA (100 μm) (for values of adenosine alone, see Methods). All values are expressed as means ± s.e.m. of 9 individual hearts

(P < 0.05 vs. previous condition (line above)).