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. Author manuscript; available in PMC: 2008 Mar 31.
Published in final edited form as: Pain. 2006 Oct 6;128(1-2):40–51. doi: 10.1016/j.pain.2006.08.023

Fig. 4.

Fig. 4

Conduction velocity properties of uninjured C fibers of the L4 spinal nerve are changed after L5 spinal nerve injury. (A) Activity-dependent slowing is enhanced after the lesion. The rCV of the last leading AP (L60) is significantly lower in the lesioned animals for both the “nociceptor” and the “non-nociceptor” population. (B) The rCV of the first trailing AP (T1) in the lesioned animals is significantly greater in the “non-nociceptor” population but significantly lower in the “nociceptor” population. (C) The rCV of the last trailing AP (T60) is significantly greater in the lesioned animals for both the “nociceptor” and the “non-nociceptor” population. All data taken from animals 8–10 days after sham or SNL surgery. (***p < 0.001; *p < 0.05; lesioned versus sham).