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. 2003 Nov 1;112(9):1342–1350. doi: 10.1172/JCI18607

Figure 6.

Figure 6

Proposed mechanism by which T cell–dependent inflammation promotes atherosclerosis but is dampened by TGF-β (a). The lack of functional TGF-β receptors on T cells abrogates the modulatory action of this cytokine, leading to aggravated atherosclerosis in E0 × CD4dnTβRII mice (b). TGF-βR, TGF-β receptor.