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. Author manuscript; available in PMC: 2008 Apr 8.
Published in final edited form as: Cell Microbiol. 2006 Sep;8(9):1430–1443. doi: 10.1111/j.1462-5822.2006.00722.x

Figure 2. P− Gc variants arise from a P+ progenitor during T84 cell infection.

Figure 2

Panel A: Percentage of cell-associated P− CFU. The pilus-dependent colony morphology of CFU arising over time in T84 cells infected with P+ FA1090 Gc was examined using a stereomicroscope. The average percentage of P− CFU ± SEM was determined for three replicate monolayers per time point. Panel B: P− Gc arise stochastically during epithelial infection. Three monolayers of T84 cells, denoted A-C, were infected with P+ FA1090. The P+ (black bars) and P− (white bars) CFU present in the cell-associated population of each monolayer at 30 h and entering the apical and basal medium from each monolayer between 27 and 30 h were enumerated as in Figs. 1A and B.