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. Author manuscript; available in PMC: 2009 Mar 20.
Published in final edited form as: Brain Res. 2008 Feb 2;1200:58–65. doi: 10.1016/j.brainres.2008.01.057

Figure 4.

Figure 4

GABAA receptor blockade in post-sensitive period slices reverts synaptic plasticity to sensitive period characteristics. Data for aCSF recordings is the same as shown in Fig. 3. (A) Mean slope of evoked field potential as a proportion of baseline recorded in the BLA before and after tetanic stimulation of LA inputs for slices from postsensitive period pups with and without added picrotoxin. Tetanic stimulation of the lateral amygdala input to basolateral amygdala induced long-term depression in post-sensitive period control slices but long-term potentiation in the presence of picrotoxin. (B) Same as A, for stimulation of coA inputs. Picrotoxin blocked the long-term depression of cortical nucleus input to basolateral amygdala seen in control post-sensitive period slices.