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. Author manuscript; available in PMC: 2008 May 2.
Published in final edited form as: Psychophysiology. 2007 Jan;44(1):98–105. doi: 10.1111/j.1469-8986.2006.00471.x

Table 1.

Biometric Model Fitting Results for the Association Between P3 Amplitude and Externalizing Disorders

Model −2lnL df Δχ2 Δdf BIC
Base model 8587.58 2074 — — —
1. ACE allow cohort differences 8630.23 2104 42.65 30 −149.2
2. ACE no cohort differences 8657.82 2113 70.24 39 −179.1
3. CE allow cohort differences 8732.21 2110 144.63 36 −85.5
4. CE no cohort differences 8762.10 2116 174.52 42 −94.0
5. AE allow cohort differences 8632.19 2110 44.61 36 −185.6
6. AE no cohort differences 8658.81 2116 71.23 42 −197.3
6a. No genetic contribution to P3-Externalizing covariance 8670.89 2117 83.31 43 −191.6
6b. P3-Externalizing covariance due to genetic factors only 8662.62 2117 75.04 43 −199.9

Note: A: additive genetic, C: shared environment, E: nonshared environment, lnL: log-likelihood, df: degrees of freedom, BIC: Bayesian information criterion. Δχ2 and Δdf are relative to the base model.