Fig. 6.

Role of FAK/Src complex in regulation of MEF2a in cardiomyocytes. (A–D) Embryonic cardiomyocytes were infected with recombinant adenoviruses encoding GFP, FAK, or its mutants, as indicated (A). In C, the cells were treated with DMSO or PP2 as indicated. Lysates were prepared and analyzed by Western blotting using α-MEF2a, α-cFAK, or α-vinculin. The intensity of the bands was quantified from three independent experiments by densitometry. The mean± SD values of relative intensity (normalized to the cardiomyocyte infected with adenovirus encoding GFP) are shown in arbitrary units (AU) in B and D. *, P < 0.05 compared with FAK and FAK plus DMSO, respectively.