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Bay 11-7082 and rapamycin suppressed bile acid–induced esophageal tumor anchorage-independent growth. A, CDCA enhanced the anchorage-independent growth abilities of SEG-1 EAC cancer cells in soft agar. B, quantitative result of A. CDCA-induced SEG-1 and BE3 EAC cancer cells’ soft agar growth ability (P < 0.05). The enhanced anchorage-independent growth could be inhibited by Bay 11-7082 and rapamycin (P < 0.001).
