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. Author manuscript; available in PMC: 2008 May 15.

Published in final edited form as: Methods Enzymol. 2008;438:367–389. doi: 10.1016/S0076-6879(07)38026-9

Fig 5 — Dilated cardiomyopathy in Rce1 ^lx/α αMyhc-Cre^+/o mice. A, Increased size of hearts from Rce1^flx/α αMyhc-Cre^+/o mice, compared with Rce1^+/+αMyhc-Cre^+/o controls. B, H&E-stained sections of an Rce1^flx/α αMyhc-Cre^+/o heart and an Rce1^+/+αMyhc-Cre^+/o heart, both from 7-month-old mice. The hearts from Rce1^flx/α αMyhc-Cre^+/o mice were invariably dilated. Note the organized left atrial thrombus (arrow) in the heart from the Rce1^flx/α αMyhc-Cre^+/o mouse. Reproduced, with permission from The Journal of Biological Chemistry (Bergo et al., 2004).