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. Author manuscript; available in PMC: 2008 Jun 2.
Published in final edited form as: J Infect Dis. 2008 Apr 1;197(7):981–989. doi: 10.1086/529203

Figure 5.

Figure 5

A and B, Inhibitory effects of monoclonal antibody (mAb) 163.5 or IgG1 isotype control mAb on T lymphocyte activation (A; *P < .002 and **P < .035) or expansion of Vβ subsets 2 and 14 (B; *P < .02) induced by Int1-negative (Int1−) C. albicans blastospores (VBIDM2) or Int1-positive (Int1+) C. albicans blastospores (DAY286). Staphylococcal enterotoxin B (SEB) served as control. Data are mean values ±2 SDs (n = 5 experiments). C and D, Inhibitory effects of mAb 163.5 or IgG1 isotype control mAb on expansion of Vβ subsets 2 and 14 (C; *P = .02 for Vβ2 and **P = .01 for Vβ14) and secretion of interferon-γ (IFN-γ) (D; *P < .01) induced by soluble Pep263 in the absence or presence of Int1− C. albicans blastospores (VBIDM2). Data are mean values ±2 SDs (n = 6 experiments).