Figure 1.

PR activity may be one mechanism by which testicular hormones masculinize neural development and subsequent adult behavior. A, Neonatal treatment of females with the PR antagonist, RU486, attenuated the masculinizing effects of testosterone propionate (TP) on the volume of the SDN-POA (27). *, Significantly different from vehicle group (P < 0.05; **, significantly different from oil-treated females (P < 0.01). B, Neonatal treatment of males with RU486 significantly reduced the percent of males displaying mounting behavior in adulthood (34). *, Significantly different from untreated and vehicle.