(A) PC-3MM2 cells were treated with siRNA targeting the 5′UTR region of EGFR mRNA, which downregulated the EGFR level after a 4-day siRNA treatment. Both WT-EGFR and kmtEGFR were re-expressed in the siRNA-treated cells and were able to upregulate SGLT1 expression. The absence of kinase activity of kmtEGFR was shown by western blotting with an anti-pEGFR antibody. β-actin served as a loading control.
(B) The re-expression of WT-EGFR and kmtEGFR also rescued the EGFR siRNA-treated cells from death. The sub-G1 proportion is indicated by the arrows. (Duplicate samples were used in each group of samples.)