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Hypothetical mechanism(s) responsible for PPARγ-dependent ERK activation and PGD2 generation by MCs. LTE4-mediated responses are MK571-sensitive and may involve both CysLT1R and an unidentified GPCR, whereas LTD4 responses are regulated by respectively positive and negative signals induced through the CysLT1R/CysLT2R heterodimer. PPRE, PPAR response element.
