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. 1999 Dec 7;96(25):14383–14388. doi: 10.1073/pnas.96.25.14383

Table 1.

Expression of the Rfz-2/β2AR chimera in Chinese hamster ovary (CHO-K) cells deficient in β-ARs and in F9 cells yields increased expression of β-AR binding, as measured by the binding of the radiolabeled, high-affinity β-adrenergic antagonist [125I]CYP

Cell line Vector [125I]CYP binding, fmol/105 cells
CHO-K EV 0.6  ±  0.2
Rfz-2/β2AR chimera 18.0  ±  2.5
F9 EV 10.3  ±  0.9
Rfz-2/β2AR chimera 20.6  ±  4.4

Data are expressed as the mean values (±SEM) of at least four different clones and two independent experiments. EV, empty vector control.