. 1999 Dec 7;96(25):14451–14458. doi: 10.1073/pnas.96.25.14451

Table 6.

Mutations lowering levels of other integral U1 or U2 snRNP proteins do not suppress snf⁺ dose effects on Sxl^M12/Y male viability

Progeny class (Cross^†)	Experimental males				Control Sxl⁺ male sibs^*, n
Progeny class (Cross^†)	Sxl allele	snRNP locus genotype	P{snf⁺} copies	Relative viability	Control Sxl⁺ male sibs^*, n
1 (A)	Sxl^M12	U1-70K⁶²/U1-70K⁶²	1	1%	71
2 (A)	Sxl^M12	+/+	0	84%	117
3 (A)	Sxl⁺	U1-70K⁶²/U1-70K⁶²	1	44%	161
4 (B)	Sxl^M12	noi²/Df(noi)	1	0%	240
5 (B)	Sxl^M12	noi²/Df(noi)	0	80%	225
6 (B)	Sxl⁺	noi²/Df(noi)	1	90%	267

For classes 3 and 6, these are U1-70K⁶²/+ and noi²/+, respectively. For all others, autosomal genotypes match experimentals. Sxl genotype was inferred from the closely linked (0.9 cM) ct marker, except that rare Sxl⁺-ct recombinants were recognized by their lack of the abdominal etching diagnostic for Sxl^M12.

^† A, w Sxl^M12ct⁶/w; U1-70K⁶²/SM6b, Cy Roi ☿☿ × ♂♂ w/Y; U1-70K⁶² P{snf⁺w^+mC}108/+. B, w Sxl^M12ct⁶/w; noi^{2{PlacW,w+mC}}/TM2, Ubx ☿☿ × ♂♂ w/Y; P{snf⁺w^+mC}108/+; Df(3R)noi-D/st Ki p^p ry.