Table 2.
Phenotypes of sna expression in different genetic background
| Maternal genotypes | Kr-lacZ embryos, +/+ or TAF/+
|
Non-Kr-lacZ embryos, TAF/+ or TAF/TAF
|
||||||
|---|---|---|---|---|---|---|---|---|
| Normal, % | Type I, % | Type II, % | n | Normal, % | Type I, % | Type II, % | n | |
| wild-type | 96.2 | 3.8 | 0.0 | 400 | ||||
| TAFII110ΔC/+ | 94.7 | 5.3 | 0.0 | 337 | ||||
| TAFII110ΔB/+ | 94.4 | 5.6 | 0.0 | 285 | ||||
| TAFII60YY/+ | 96.3 | 3.7 | 0.0 | 351 | ||||
| dl/+; TAFII110ΔC/+ | 23.2 | 57.1 | 19.7 | 452 | 8.1 | 55.1 | 36.8 | 494 |
| dl/+; TAFII110ΔB/+ | 22.4 | 63.0 | 14.6 | 343 | 6.9 | 64.8 | 28.3 | 361 |
| dl/+; TAFII60YY/+ | 21.7 | 65.0 | 13.3 | 309 | 11.8 | 58.1 | 30.1 | 356 |
Normal, Type I, and Type II are as in Table 1. Wild-type females or female flies with designated genotype were mated with wild-type males or male flies with the corresponding TAFIImutations over the Kr-lacZ-marked chromosome. Embryos with Kr-lacZ expression were either wild-type or contained one mutant copy of the indicated TAF gene; embryos without Kr-lacZ expression contained either one copy of mutant TAFs or were homozygous for the TAF mutation.