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. 2008 Jul 22;105(30):10279–10284. doi: 10.1073/pnas.0805284105

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© 2008 by The National Academy of Sciences of the USA

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Fig. 5. — Blockade of β-ADr and NMDAr in dorsal CA1 does not affect the biochemical changes induced by IA training in BLA, and blockade of β-ADr and NMDAr in BLA does not affect the biochemical changes induced by IA training in dorsal CA1. (A) Representative immunoblots show the lack of effect of the intra-CA1 infusion of TIM and AP5 on the increase in ERK2 (pERK2; t = 0.95, P > 0.1 for TIM; t = 0.27, P > 0.1 for AP5; n = 4 per group) and TH (pTH; t = 0.16, P > 0.1 for TIM; t = 1.19, P > 0.1 for AP5; n = 4 per group) phosphorylation induced in BLA by IA training. (B) Representative immunoblots show the effect of the intra-CA1 administration of TIM and AP5 on the increase in CaMKII (pCaMKII; t = 0.24, P > 0.1 for TIM; t = 5.68, P < 0.01 for AP5; n = 4 per group) and GluR1 (pGluR1; t = 0.12, P > 0.1 for TIM; t = 3.54, P < 0.05 for AP5; n = 4 per group) phosphorylation induced in that region by IA training. (C) Representative immunoblots show the lack of effect of the intra-BLA infusion of TIM and AP5 on the increase in pCaMKII (t = 0.84, P > 0.1 for TIM; t = 0.40, P > 0.1 for AP5; n = 4 per group), pGluR1 (t = 0.03, P > 0.1 for TIM; t = 0.34, P > 0.1 for AP5; n = 4 per group), and pERK2 (t = 0.32, P > 0.1 for TIM; t = 0.66, P > 0.1 for AP5; n = 4 per group) phosphorylation induced in the dorsal CA1 region by IA training.