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. 2008 May 2;295(1):H69–H76. doi: 10.1152/ajpheart.00341.2008

Fig. 1.

Fig. 1.

A: pressure-passive diameter relationship of mesenteric resistance arteries from control (sham), hypertensive (HT), Type 1 diabetic (D1), HT + transforming growth factor (TGF)-β1 antibody (TGF-β1-AB), and D1 + TGF-β1 antibody (TGF-β1-AB) mice. *P < 0.05 statistically significant differences between sham vs. HT; sham vs. D1, HT vs. HT + TGF-β1-AB; and D1 vs. D1 + TGF-β1-AB. B: Western blot analysis and cumulative data showing the increased collagen type 1 of mesenteric resistance arteries from control (sham), hypertensive (HT), Type 1 diabetic (D1), HT + TGF-β1-AB, and D1 + TGF-β1-AB mice. *P < 0.05 statistically significant differences between: sham vs. HT; sham vs. D1; HT vs. HT + TGF-β1-AB; and D1 vs. D1 + TGF-β1-AB; n = 7 mice for each experiments.

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