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. 1997 Dec 23;94(26):14596–14601. doi: 10.1073/pnas.94.26.14596

Table 2.

Transduction by rKmK donors

Donor* Recipient Percent of transductants with donor phenotypes
DnaC+ rKmK DnaC+ TetS
NM854 NM815 (rK+mK+) 52 96
NM854 NM814 (rKmK+) 50 96
NM840 NM815 (rK+mK+) 43 96
NM840 NM814 (rKmK+) 56 94
NM679 NM815 (rK+mK+) 98 (94) 96 (90)
NM679 NM814 (rKmK+) 100 (95) 97 (92)

The order of genes on the chromosome is hsdS hsdM hsdR dnaC Tn10. Cotransduction frequencies are based on a minimum of 50 colonies. 

*

See Table 1 for identification of hsd deletion. The hsdSM deletion in NM854 is tagged by a gene conferring resistance to kanamycin. The other deletions were recognized by their restriction and modification phenotype. 

Donor strain for data within brackets was without λhsdM+S+ prophage and therefore mK