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Lack of effect of resistin deficiency on putative mediators of hepatic insulin resistance. WT and RKO mice were fed a high-fat diet for 10 wk, and ob/ob and ob/ob-RKO mice were fed normal chow (A–C). Data are means ± SE arbitrary densitometric units. Suppressors of cytokine signaling (SOCS)-3 was normalized to β-actin, and phosphorylated (p)-AMP-activated protein kinase (AMPK) was normalized to total AMPK (n = 3/group).
