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. Author manuscript; available in PMC: 2009 Jul 15.

Published in final edited form as: Biochem Pharmacol. 2008 May 8;76(2):198–207. doi: 10.1016/j.bcp.2008.05.004

Fig. 1 — A: Effect of DHCB on distribution of F-actin and tubulin. Hke-3 cells were left untreated (CTRL) or were left untreated or were treated with DHCB (10 mM) for 4 or 12 hours. Immunofluorescence was performed with antibodies specific for tubulin (green) or by staining with phalloidin for F-actin (red). B: Inhibition of S phase and induction of G0/G1 and G2/M arrest in cucurbitacin treated cells. HCT116 and Hke-3 cells were left untreated or were treated with 12 µM or 25 µM DHCB for 12 hours and the cell cycle profile was determined by PI staining.

Fig. 1 — A: Effect of DHCB on distribution of F-actin and tubulin. Hke-3 cells were left untreated (CTRL) or were left untreated or were treated with DHCB (10 mM) for 4 or 12 hours. Immunofluorescence was performed with antibodies specific for tubulin (green) or by staining with phalloidin for F-actin (red). B: Inhibition of S phase and induction of G0/G1 and G2/M arrest in cucurbitacin treated cells. HCT116 and Hke-3 cells were left untreated or were treated with 12 µM or 25 µM DHCB for 12 hours and the cell cycle profile was determined by PI staining.

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