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. 2008 Sep 1;205(9):2111–2124. doi: 10.1084/jem.20072076

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© 2008 Das et al.

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.jem.org/misc/terms.shtml). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).

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Figure 8. — Depletion of serum l-arginine is associated with elevated serum and liver arginase activity during periods of HBV disease activity in patients with CHB. (a) Serum l-arginine levels were compared in healthy donors (n = 21), patients with low-to-moderate liver inflammation (ALT < 100 IU/liter; n = 13), and patients with high liver inflammation (ALT > 100 IU/liter; n = 11; control vs. high ALT P < 0.01; low ALT vs. high ALT P < 0.05). (b) Graphs depict longitudinal fluctuations in serum arginase activity (dashed line, IU/liter) in association with viral load (solid gray line, HBV DNA, IU/ml) and ALT (shaded histogram, IU/liter) for two patients with flares of CHB. (c) The arginase activity of liver homogenates is shown for five individuals with CHB and five individuals with nonviral liver pathology.

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