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. Author manuscript; available in PMC: 2008 Sep 8.
Published in final edited form as: Biochemistry. 2006 Jan 31;45(4):1242–1254. doi: 10.1021/bi051669r

Table 2.

ANS Fluorescence in the Presence of WT and Mutant Forms of Lipid-Free apoA-I and “Reference Proteins”. Trp Fluorescence of WT and Mutant Forms of Lipid-free apoA-I.

Protein WMF c (nm) I c (relative units d) I c (relative units e) WMF f (nm)
WT b 479 (-37)c 4.5 1.0 335
apoA-I[D102A/D103A]b 475 (-41) 6.3 1.3 338
apoA-I[R116V/ K118A]b 479 (-37) 4.0 0.9 336
apoA-I[R160V/H162A]b 475 (-41) 6.5 1.4 337
apoA-I[Δ(61-78)]b 476 (-40) 7.0 1.6 *





WT a 480 (-36) 4.3 1.0 335
apoA-I[E110A/E111A] a 477 (-39) 6.8 1.5 337
apoA-I[Δ(121-142)] a 492 (-24) 3.3 0.7 335





Bovine Serum Albumin 475 (-41) 9.5 2.1 N/A
Carbonic Anhydrase 516 1.0 0.2 N/A
ANS in buffer alone 516 1.0 0.2 N/A
a

Proteins were expressed in the adenovirus

b

Proteins were expressed in the baculovirus expression system.

c

ANS fluorescence in the presence of 0.05 mg/ml of WT or the mutant forms of apoA-I, carbonic anhydrase or bovine serum albumin, or in PBS buffer alone. Values in parentheses show changes in WMF compared to that for ANS in PBS buffer.

d

ANS fluorescence intensity in relative units compared to the fluorescence in the buffer alone.

e

ANS fluorescence intensity in relative units compared to the fluorescence in the presence of WT expressed in the baculovirus expression system.

f

Intrinsic Trp fluorescence of WT and mutant apoA-I in PBS.

*

WMF for the apoA-I[Δ(61-78)] was not determined because of the lack of Trp72 in this mutant.