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. 2007 Jul 23;100(3):651–657. doi: 10.1093/aob/mcm136

Fig. 3.

Fig. 3.

Phenotypic effects and expression of DL in leaves and other organs in a proportion of proCRC::DL transformants. (A) A mature plant transformed with proCRC::DL (corresponding to plant 1 in Fig. 3G), showing normal development of leaves and inflorescence architecture. (B) A seedling transformed with proCRC::DL, showing aberrant leaf development. (C) A mature plant transformed with proCRC::DL (corresponding to plant 9 in Fig. 3G), showing aberrant leaf development and plant architecture. (D) The underside of a leaf from the proCRC::DL transformant shown in (C). (E) A pro35S::CRC transformant showing a leaf phenotype similar to that of some proCRC::DL transformants. (F) Mature rosette leaves, showing the effect of overexpression of CRC and DL transgenes in leaf tissue. (G) Northern hybridization of a DL cDNA probe to RNA from leaves and inflorescences of three proCRC::DL transformants (plants 1, 3 and 5) that showed normal leaf development, and to RNA from leaves of three proCRC::DL transformants (plants 9, 13 and 33) that showed aberrant leaf development. High levels of DL expression in leaves correlate with aberrant leaf phenotypes.