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. 2008 Sep 22;182(6):1083–1097. doi: 10.1083/jcb.200803174

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© 2008 Brown et al.

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.jcb.org/misc/terms.shtml). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).

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Figure 9. — A model for involvement of splicing factor aggregations in the stochastic association of transcriptionally active genes. Genes in the process of transcriptional elongation accumulate splicing-related factors (left). Nuclear speckles are known to form as concentrations of such factors in early G1 and randomly during interphase. This process of nucleation may pull more mobile chromatin (around gene A) into association with other active genes (gene B; right).