. Author manuscript; available in PMC: 2008 Sep 23.

Published in final edited form as: J Cereb Blood Flow Metab. 2006 Aug 2;27(4):729–740. doi: 10.1038/sj.jcbfm.9600376

Table 2.

Regional loss of Nissl staining intensity post HI injury

Dietary groups and HI subgroups	Hippocampal subarea^a
	CA1	CA3ab	CA3c	Dentate gyrus	Parietal cortex
Iron-sufficient
15 min (n = 6)	3.3 ± 4.2	0.0 ± 2.3	0.0 ± 2.3	0.3 ± 1.1	0.9 ± 0.2
30 min (n = 6)	0.6 ± 6.2	0.0 ± 2.6	4.8 ± 5.4	0.0 ± 4.3	0.6 ± 0.2
45 min (n = 6)	6.8 ± 7.1	0.0 ± 2.1	0.1 ± 3.0	1.0 ± 1.9	0.4 ± 0.3
Iron-deficient^b
15 min (n = 6)	3.7 ± 4.9	0.0 ± 1.2	0.0 ± 3.3	7.8 ± 4.6	0.2 ± 0.1
30 min (n = 9)	18.8 ± 3.0^c	6.8 ± 6.9	2.5 ± 3.7	2.6 ± 2.3	0.3 ± 0.2
45 min (n = 8)	17.6 ± 6.1	2.1 ± 3.9	0.0 ± 2.9	1.8 ± 2.3	2.8 ± 1.7

Values = mean ± s.e.m. % loss of Nissl staining intensity on the HI hemisphere when compared with the corresponding brain region on the control hemisphere.

Nomenclature based on Johansen (1993).

Main effect of iron-deficiency on Nissl staining intensity loss was seen for subarea CA1 (P = 0.03, univariate ANOVA).

Significantly different between iron-sufficient and iron-deficient groups (P < 0.01, Bonferroni-adjusted t-test).