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. Author manuscript; available in PMC: 2009 Oct 1.
Published in final edited form as: J Cell Physiol. 2008 Oct;217(1):207–214. doi: 10.1002/jcp.21495

Fig. 8.

Fig. 8

P47phox deficiency attenuates IRF1 and AP1 activation. Wild type and p47phox-deficient (p47phox−/−) endothelial cells were incubated with or without LPS+IFNγ for 2 h. Subsequently the nuclear proteins were extracted for determination of the DNA binding activities of NFκB, IRF1 and AP1 by EMSA. Shown are representative EMSA results and the summaries of band intensities (expressed in arbitrary units; mean ± SEM values for three independent experiments). *Significant difference from the vehicle treated wild type group (P < 0.05). #Significant difference from the LPS+IFNγ-stimulated wild type group (P < 0.05).