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. 2008 Sep 23;4(6):338–344. doi: 10.7150/ijbs.4.338

Table 1.

Potential SOS boxes of genes that bind or do not bind LexA repressor.

Gene SOS box sequence HI* NM**
Consensus TACTGTATATATATACAGTA 0
A. Genes whose SOS boxes bind LexA and are regulated by LexA represor
recA TACTGTATGAGCATACAGTA 4.31 1
umuDC TACTGTATATAAAAACAGTA 2.77 2
uvrB AACTGTTTTTTTATCCAGTA 6.11 5
polB GACTGTATAAAACCACAGCC 12.09 5
lexA1 TGCTGTATATACTCACAGCA 6.34 4
lexA2 AACTGTATATACACCCAGGG 8.32 6
B. Genes whose potential SOS boxes do not bind LexA but re not LexA regulated
intE GGCTGCTGAAAAATACAGAA 16.04 7
ymfI TTCTGTACCAGAAAACAGTT 15.48 8
ymfM AGCTGCAGGAGCATGCAGCA 19.32 3
lit TGATGACAGAGTGTCCAGTG 20.32 8
C. Genes whose SOS boxes do not bind LexA in spate of low HI value
yigN AACTGGACGTTTGTACAGCA 9.27 5
dinJ AGCTGAATAAATATACAGCA 7.06 3

Potential SOS boxes (sequence on coding strand) that bind (A), or do not bind (B and C) LexA repressor.

HI* denotes heterology index; NM** denotes the number of mismatches in SOS boxes deviating from a perfect palindrome. The lack of LexA repressor binding despite a relatively low HI value (section C) testifies that there is no direct correlation between them 28. Anyhow, it indicates that the HI value cannot be the only indicator of the ability of an SOS box to bind LexA. The number of mismatches in the palindromic SOS boxes in each of the sections is similar, and does therefore not determine LexA binding ability with the SOS boxes. Data in parts A and C are from ref. 28, those in part B are from ref. 31.