Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2008 Oct 6;183(1):11–17. doi: 10.1083/jcb.200807052

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2008 Jang et al.

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.jcb.org/misc/terms.shtml). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).

PMC Copyright notice

Figure 3. — Sln enhances LKB1-dependent apoptosis without affecting cell cycle progression or cell size. (A) Immunostaining against BrdU (top), phosphohistone H3 (middle), or Elav (bottom) in larval eye discs from the indicated genotypes. Arrowheads mark the second mitotic wave posterior to the morphogenetic furrow. Posterior at right. (B) Cytoplasmic GFP-marked flipped-out mitotic clones expressing LKB1 and Sln in larval fat body (top), wing disc (bottom left), or eye disc (bottom right). Filamentous actin was stained in fat body (top, red). Nuclear GFP is nonspecific autofluorescence in fat body (top). The time after clonal induction is indicated (bottom). (C and D) TUNEL (red) and AO (green) staining in larval eye discs from the indicated genotypes. The penetrances of apoptosis phenotypes were 100% (n = 10). Posterior at right. Bars, 50 μm.