Table 1.
Analysis of FLP/FRT-induced ter94 mutant clones in somatic tissue
| Genotype | HS | NHS | ||
|---|---|---|---|---|
| A | P[hsFLP] ; P[FRT] l(2)k15502 | 86% | 65% | |
| + P[FRT] P[arm-LacZ] | n = 179 | n = 156 | ||
| B | ±; P[FRT] l(2)k15502 | 11% | 14% | |
| Y P[FRT] P[arm-LacZ] | n = 106 | n = 154 | ||
| C | P[hsFLP] ; P[FRT] [ovoD1] | 13% | 4% | |
| + P[FRT] P[arm-LacZ] | n = 106 | n = 77 | ||
| D | ±; P[FRT] [ovoD1] | 2% | 8% | |
| Y P[FRT] P[arm-LacZ] | n = 53 | n = 50 | ||
Appropriate crosses were established to produce the progeny shown. Note that only females carry the transposon encoding the Flipase recombinase (e.g. [hsFLP]). The l(2)k15502 allele of the ter94 gene was used for production of mitotic clones. The percentage of animals of a given genotype that were missing at least one bristle was scored. P[FRT] is P[FRT; w+]2R-G13, l(2)k15502 is a lethal P-allele of ter94; [ovoD1] is P[ovoD1; w+]. P[hsFLP]; P[FRT; w+]2R-G13/P[FRT; w+]2R-G13 P[ovoD1; w+].