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. 2008 Oct 10;105(42):16392–16397. doi: 10.1073/pnas.0803997105

Fig. 7.

Fig. 7.

AS2 represses KAN1 and KAN2. (A–F) The expression of pKAN1:GUS in the leaf primordia of 3-day-old (A–C) and 9-day-old (D–F) seedlings. (A and D) Wild type. (B and E) as2–5D. (C and F) as2–1. Cotyledons were removed to reveal the leaf primordia. The arrow in D indicates abaxial GUS expression in the petiole. The expression domain of pKAN1:GUS is reduced by the gain-of-function as2–5D mutation (B and E) and expanded by the loss-of-function as2–1 mutation (C and F). (G–N) The rosette (G–J) and fifth leaf (K–N) of 18-day-old plants. (G and K) kan1–11. (H and L) as2–5D. (I and M) kan1–11 as2–5D. (J and N) kan1–11 kan2–5. Adaxial, abaxial, and side views of the leaf are shown from left to right. The phenotype of kan1–11 as2–5D resembles that of kan1–11 kan2–5. (Scale bars: A–C, 20 μm; D–F, 100 μm; G–N, 0.5 cm.)