Skip to main content
. Author manuscript; available in PMC: 2009 Oct 1.
Published in final edited form as: Mol Microbiol. 2008 Jun 28;70(1):3–14. doi: 10.1111/j.1365-2958.2008.06333.x

Fig. 1.

Fig. 1

Schematic representation of RIVET analysis of B. pertussis iron transport system genes. Transcriptional activity of alcA (alcaligin), bfeA (enterobactin) and bhuR (heme) iron system promoters (designated PIron) in vivo drives production of the site-specific resolvase TnpR, which catalyzes excision of the linked kanamycin resistance marker (neo) by recombination between the res1 sites. Loss of kanamycin resistance serves as a stable heritable marker of prior gene expression.