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. 2008 Oct;59(14):4045–4058. doi: 10.1093/jxb/ern245

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© 2008 The Author(s).

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/2.0/uk/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

This paper is available online free of all access charges (see http://jxb.oxfordjournals.org/open_access.html for further details)

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Fig. 7. — Effects of 3,4-DHP on the development of the cotyledon vasculature in tobacco. (A) The control cotyledon vasculature contained a main vein (indicated by an arrow) and small branched veins. (B) The vasculature contained two main veins (indicated by arrows) and small branched veins in the single cotyledon which originates from two cotyledons incompletely fused when treated with 100 μM 3,4-DHP. (C) The vasculature of another single cotyledon, in this situation, only one of the cotyledon primordia could develop into a normal cotyledon, therefore the vasculature was similar to the control, which contained a main vein (indicated by an arrow) and small branched veins. (D) The vasculature in two asymmetric cotyledons induced by 100 μM 3,4-DHP. The larger cotyledon has three main veins (indicated by three arrows), but the smaller cotyledon has a similar vasculature to the control. (E) In a cup-shaped cotyledon induced by 100 μM 3,4-DHP, the vasculature contained more than two main veins (indicated by arrows) and small branched veins. (F) The vasculature in the three cotyledon seedling induced by 100 μM 3,4-DHP contained a main vein (indicated by an arrow) and small branched veins. Bar=1 mm.