Skip to main content
. Author manuscript; available in PMC: 2008 Nov 3.
Published in final edited form as: J Biol Chem. 2006 Dec 1;282(7):5026–5036. doi: 10.1074/jbc.M609360200

Figure 2.

Figure 2

(A) Morphological comparison of male internal reproductive organs in wild-type (a) and FKBP52-deficient adult mice (b). FKBP52-deficient males have overall normal testis (Tes) formation and normal epididymis (Epi), but significantly smaller seminal vesicles (SV). Kid: kidney; Bl: bladder; Pe: penis; Black arrow indicates urethral opening. (B) Histological analysis of prostate gland development in FKBP52-deficient and age-matched littermate control mice at birth (P0) to 3 month old. Prostate glands are initially formed during embryonic developmental in both wild-type (a) FKBP52-deficient mutant (b), but lack further growth after the birth (d) and eventually become dysgenic in FKBP52-deficient adult males (f) compared to littermate wild-type mice (c and e). Prostate glands are indicated by black arrows.