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. 2008 Sep 9;35(3):305–315. doi: 10.1152/physiolgenomics.90248.2008

Fig. 2.

Fig. 2.

Matrix of modules related to myocardial infarction (MI) and pressure-induced overload. This figure presents the 266 modules that had their expression change in the clinical conditions of MI and pressure-induced overload. The figure presents the sample attributes directly related to MI and pressure-induced overload (banding) such as MI infarcted region or banding after 48 h. We also present additional sample attributes that represent a variety of clinical conditions, including embryonic stages, transgenic causes of heart stress and failure (CSQ transgenic, MLP knockout, G4D transgenic), transgenic activation of TNF-α or three MAP kinases (ERK, p38, and JNK). Each row in this matrix represents a module; each column represents a sample attribute. Each cell represents the percentage of samples that were induced (red) or repressed (green) samples out of all samples belonging to one specific module-attribute combination. Color intensity corresponds to the size of the percentage, where black represents zero and indicates that the amount of samples with this attribute that were repressed or induced was not statistically significant. Concise biological labels were added (colored bars, right side), and modules are grouped by these labels.