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. Author manuscript; available in PMC: 2008 Nov 24.
Published in final edited form as: Arch Neurol. 2008 Jul;65(7):906–912. doi: 10.1001/archneur.65.7.906

Table 3.

Results of cross-sectional and longitudinal analyses

Temporal Cingulate
Aβ40 Aβ42 Aβ42
r p r P r p
Blessed DRS prior to death Tris −0.040 0.854 0.252 0 234 0.042 0.845
Triton 0.485 0.016 −0.185 0.388
SDS 0.439 0.032 0.168 0.432
FA −0.075 .728 −0.065 0.763 −0.189 0.376
Illness Duration Tris 0.567 0.002 0.130 0.518 0.117 0.568
Triton 0.286 0.149 −0.039 0.848
SDS −0.087 0.667 0.199 0.330
FA 0.510 0.007 −0.097 0.629 0.004 0.986
F p F P F p
ApoE Tris 5.154 0.014 0.226 0.799 0.508 0.608
Triton 0.874 0.430 0.603 0.556
SDS 1.272 0.299 0.087 0.917
FA 9.343 0.001 1.695 0.205 0.278 0.760
β p β P β p
Rate of cognitive decline Tris −1.237 0.268 0.005 0.997 2.070 0.145
Triton 1.750 0.200 −0.112 0.940
SDS 3.506 <0.001 −2.747 0.011
FA −2.106 0.070 1.019 0.375 −2.477 0.020

All analyses reflect unadjusted models using raw data, except Blessed correlations which include time from last evaluation until death as a covariate. The reported β in the GEE analysis is that of the time × Aβ interaction term; positive values indicate a more rapid cognitive decline for subjects with measured Aβ in the upper median.